nature.com

A molecule designed to activate iron locked up in organelles called lysosomes and thereby induce cell death might offer a way to tackle treatment-resistant cancer.

Chronic stress response activation impairs cell survival and causes devastating degenerative di-seases 1-3. Organisms accordingly deploy silencing factors, such as the E3 ubiquitin ligase SIFI, to terminate stress response signaling and ensure ...

Contact has not been made.

3D structure of the tongue's sweet-sensing protein could guide future food designs.

Approach versus avoidance responses are driven by D1 and D2 neurons in the ventral hippocampus.

Introducing spin-orbit coupling by substrate proximity effect leads to an enhancement of superconducting phases in rhombohedral trilayer graphene.

At sufficiently high concentrations, some of the compounds called per- and poly-fluoroalkyl substances self-organize into two-layered membranes.

A technique called condense-seq has been developed to measure nucleosome condensability and used to show that mononucleosomes contain sufficient information to condense into large-scale compartments without requiring any external factors.

A critically low abundance of older herring due to age-selective fisheries resulted in an approximately 800-km poleward shift in main spawning.

Some cancer cells exhibit high loads of reactive iron in lysosomes, and this feature is exploited by using fentomycin-1, a newly developed small molecule, to induce ferroptosis.